The research discovered that kids who'd indications suggesting they might be developing type 1 diabetes had significantly enterovirus diseases occurring at least a year before.
That means the entire body's immune system destroys healthy insulin-producing cells called islet cells. The cells that attack the healthy cells of the body's are called autoantibodies, and you can find particular autoantibodies for type 1 diabetes. Before the indications of type 1 diabetes start these autoantibodies appear.
A constantly working insulin pump or multiple daily injections are required to replace that lost insulin.
About 5 percent of people who have diabetes have type 1.
She is a researcher in the University of Tampere in Finland.
"This autoimmune process appears to begin several months following the disease, indicating that slowly operating mechanisms are involved," she added.
The researchers stressed this study was not designed to discover a cause and effect relationship.
"Nevertheless, the collecting evidence certainly indicates an organization exists between both of these disorders," said the study's senior author, Dr. Heikki Hyoty. He is a professor of virology in the University of Tampere.
"It's probable that enterovirus disease alone cannot cause diabetes, but nevertheless, it may do so in some genetically susceptible people," he said.
Researchers examined more than 1,673 feces group control samples from the instance kids, and more than 3,100 from the
They discovered diseases in 169 and 108 instance kids in the control group.
The study team also noticed the surplus of diseases in the instance kids happened more than 12 months ahead of the initial positive autoantibody was seen.
"It's plausible that this kind of interim interval exists since it takes time ahead of the virus could activate immune mechanisms which could bring about the autoimmune process," said Honkanen.
There is no known solution to stop enteroviruses — except for enterovirus 71, for which there are vaccines and polio, the study authors said. However, this study, in addition to evidence that is previous, indicate that vaccines for other enteroviruses might lessen the prevalence of the symptoms of diabetes.
"Nevertheless, the development of this type of vaccine for human use is a long procedure," Hyoty described.
Jessica Dunne is manager of discovery research at JDRF (once the Juvenile Diabetes Research Foundation).
But, she agreed using the study writers that enteroviruses are likely not the sole environmental factor in the creation of type 1 diabetes. "Type 1 diabetes is a clinical investigation, also it is likely that individuals get there from multiple pathways. It may not always be enteroviral diseases," she said.